This research investigates whether zinc plays a critical role in the ability of psychedelic drugs to reopen social reward critical periods in the brain. Using mouse models, the study examines how zinc influences social behavior following psychedelic treatment, potentially revealing mechanisms of brain plasticity relevant to autism, social anxiety, and social connection.

This research uses cavefish to reveal how evolution reshapes the brain. By comparing surface and cave-adapted forms, it shows that neural circuits lost to vision are repurposed for touch and smell. These findings demonstrate how evolution refines existing brain structures to meet environmental demands.

Mental health disorders disrupt neural connections in the brain, yet most treatments only manage symptoms. This research explores psychedelic-inspired drugs that restore lost brain connections without hallucinogenic effects, using automated imaging tools to identify compounds that rebuild neural structure and offer lasting recovery.