This research shows that pauses in information streams alter decision-making. After a break, the brain increases effort, giving greater weight to subsequent information—a “peak-after-break” effect. A computational model explains this as a performance-effort tradeoff. Findings challenge traditional theories and suggest strategic pauses can shape attention, memory, and judgment.

In our complex world, how do humans learn and make decisions when their cognitive resources are limited? My thesis introduces a new theory called "policy compression" to answer this question! The basic idea is that people simplify their decision-making processes to reduce the mental effort required, without significantly compromising the benefits or rewards of those decisions. I use computational modeling, human experiments, and brain studies in rats to explain why people exhibit certain decision-making patterns, like the tendency to stick with familiar choices, and why they use strategies like "chunking" to reduce mental load. I also propose that different brain regions work together to balance mentally taxing decisions with more automatic, habitual decisions. This allows the brain to optimize behavior in complex environments. In conclusion, my thesis offers a new way to understand how humans and animals make decisions with limited mental resources, and shows how the brain organizes itself to handle decision-making efficiently.

This PhD uses brain-inspired AI to decode vision from neural data. Using human fMRI (24 hours of Doctor Who) and monkey electrophysiology, signals are transformed into 2D brain maps to improve reconstruction. The model learns receptive-field structure, compares contributions of V1/V4/IT, and aims for efficient, interpretable decoding with applications to neuroscience and BCIs.

This research explores how immune-related cells and molecules, beneficial in wound healing, may become harmful in Parkinson’s disease. Using the fruit fly as a model organism, the study investigates which inflammatory processes contribute to brain damage. Early results suggest that excessive activation worsens degeneration, offering potential targets for future therapies.