This research investigates whether zinc plays a critical role in the ability of psychedelic drugs to reopen social reward critical periods in the brain. Using mouse models, the study examines how zinc influences social behavior following psychedelic treatment, potentially revealing mechanisms of brain plasticity relevant to autism, social anxiety, and social connection.

This research compares three experimental models of anxiety — threat of shock, CO₂-induced panic, and speech-induced social anxiety — to reveal how each affects cognition. Findings show distinct patterns in attention, memory, and loss aversion, offering insights that could improve and better tailor treatments for anxiety disorders.